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To view or add a comment, sign in Transplant vasculopathy Minimizing transplant vasculopathy in a renal transplant recipient is addressed by controlling the major drivers of accelerated atherosclerosis and endothelial dysfunction: calcineurin inhibitor (CNI)associated hypertension steroid-associated dyslipidemia, and mammalian target of rapamycin (mTOR) inhibitorassociated dyslipidemia

Parallel randomized studies in Hashimoto thyroiditis and Graves disease should pre-specify metabolic co-endpoints (Homeostatic Model Assessment for Insulin Resistance (HOMA-IR) and Magnetic Resonance Imaging-Proton Density Fat Fraction (MRI-PDFF) for liver fat) to test the immunometabolic hypothesis suggested by observational signals and secondary analyses [32,60]