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Inhibiting the activation of mitochondrial phagocytosis by inhibiting either the PINK or PRKN signaling pathways led to the increased production of ROS and activation of inflammasome in small airway epithelial cells of patients with COPD (Ito et al., 2015)

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Conversely, the expression of carbohydrate kinase-like protein (CARKL) was inhibited upon the activation of the M1 phenotype, which under basal conditions supports the PPP