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Due to the development of 12-hydroxyeicosatetraenoic acid (12-HETE) and 13-hydroxy octadecadienoic acid (13-HODE), which are peroxidation products of the liver, small intestine, and plasma, exposure to ultrafine particles, like diesel, might exacerbate inflammation and oxidative stress in these areas (171, 172)
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Sailors who decided to try smoking the tobacco soon found it became a habit
Conclusions Our findings suggest that in utero tobacco exposure, early-life smoking, and environmental pollution independently and jointly increase the incidence of COPD
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